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A common genetic variant of a mitochondrial RNA processing enzyme predisposes to insulin resistance
Journal article   Open access   Peer reviewed

A common genetic variant of a mitochondrial RNA processing enzyme predisposes to insulin resistance

G. Rossetti, J.A. Ermer, M. Stentenbach, S.J. Siira, T.R. Richman, D. Milenkovic, K.L. Perks, L.A. Hughes, E. Jamieson, G. Xiafukaiti, …
Science Advances, Vol.7(39), Art. eabi7514
2021
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Abstract

Mitochondrial energy metabolism plays an important role in the pathophysiology of insulin resistance. Recently, a missense N437S variant was identified in the MRPP3 gene, which encodes a mitochondrial RNA processing enzyme within the RNase P complex, with predicted impact on metabolism. We used CRISPR-Cas9 genome editing to introduce this variant into the mouse Mrpp3 gene and show that the variant causes insulin resistance on a high-fat diet. The variant did not influence mitochondrial gene expression markedly, but instead, it reduced mitochondrial calcium that lowered insulin release from the pancreatic islet β cells of the Mrpp3 variant mice. Reduced insulin secretion resulted in lower insulin levels that contributed to imbalanced metabolism and liver steatosis in the Mrpp3 variant mice on a high-fat diet. Our findings reveal that the MRPP3 variant may be a predisposing factor to insulin resistance and metabolic disease in the human population.

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Source: InCites

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Collaboration types
Domestic collaboration
International collaboration
Citation topics
1 Clinical & Life Sciences
1.197 Molecular & Cell Biology - Mitochondria
1.197.564 Mitochondrial Function
Web Of Science research areas
Biochemistry & Molecular Biology
ESI research areas
Molecular Biology & Genetics
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