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Dengue-specific CD8+ T cell subsets display specialized transcriptomic and TCR profiles
Journal article   Open access   Peer reviewed

Dengue-specific CD8+ T cell subsets display specialized transcriptomic and TCR profiles

Y. Tian, M. Babor, J. Lane, G. Seumois, S. Liang, N.D.S. Goonawardhana, A.D. De Silva, E.J. Phillips, S.A. Mallal, R. da Silva Antunes, …
Journal of Clinical Investigation, Vol.129(4), pp.1727-1741
2019
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Abstract

Accumulating evidence demonstrates that CD8+ T cells contribute to protection from severe dengue virus (DENV) disease and vaccine efficacy. Nevertheless, molecular programs associated with DENV-specific CD8+ T cell subsets have not been defined. Here, we studied the transcriptomic profiles of human DENV-specific CD8+ T cells isolated after stimulation with DENV epitopes from donors who had been infected with DENV multiple times and would therefore be expected to have significant levels of adaptive immunity. We found that DENV-specific CD8+ T cells mainly consisted of effector memory subsets, namely CD45RA−CCR7− effector memory (Tem) and CD45RA+CCR7− effector memory re-expressing CD45RA (Temra) cells, which enacted specific gene expression profiles upon stimulation with cognate antigens. DENV-specific CD8+ T cell subsets in general, and Temra cells in particular, were fully activated and polyfunctional, yet associated with relatively narrow transcriptional responses. Furthermore, we found that DENV-specific CD8+ Tem and Temra cells showed some unique T cell receptor features in terms of overlap and variable (V) gene usage. This study provides a transcriptomic definition of DENV-specific activated human CD8+ T cell subsets and defines a benchmark profile that vaccine-specific responses could aim to reproduce.

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Collaboration types
Domestic collaboration
International collaboration
Citation topics
1 Clinical & Life Sciences
1.228 Virology - Tropical Diseases
1.228.200 Mosquito-borne Viruses
Web Of Science research areas
Medicine, Research & Experimental
ESI research areas
Clinical Medicine
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