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Renal pathology of polycystic kidney disease and concurrent hereditary nephritis in Bull Terriers
Journal article   Peer reviewed

Renal pathology of polycystic kidney disease and concurrent hereditary nephritis in Bull Terriers

C.A. O'Leary, M. Ghoddusi and C.R. Huxtable
Australian Veterinary Journal, Vol.80(6), pp.353-361
2002
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Abstract

Objective To describe the renal lesions in Bull Terrier poly-cystic kidney disease (BTPKD), to confirm that the renal cysts in BTPKD arise from the nephron or collecting tubule, and to identify lesions consistent with concurrent BTPKD and Bull Terrier hereditary nephritis (BTHN). Design Renal tissue from five Bull Terriers with BTPKD and eight control dogs was examined by light and transmission electron microscopy. Clinical data were collected from all dogs, and family history of BTPKD and BTHN for all Bull Terriers. Results In BTPKD the renal cysts were lined by epithelial cells of nephron or collecting duct origin that were usually squamous or cuboidal, with few organelles. They had normal junctional complexes, and basal laminae of varying thicknesses. Glomeruli with small, atrophic tufts and dilated Bowman's capsules, tubular loss and dilation, and interstitial inflammation and fibrosis were common. Whereas the lesions seen in BTHN by light microscope were nonspecific, the presence of characteristic ultrastructural glomerular basement membrane (GMB) lesions and a family history of this disease indicated concurrent BTHN was likely in three of five cases of BTPKD. Conclusion This paper provides evidence that renal cysts in BTPKD are of nephron or collecting duct origin. In addition, GBM lesions are described that strongly suggest that BTPKD and BTHN may occur simultaneously.

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Collaboration types
Domestic collaboration
Citation topics
1 Clinical & Life Sciences
1.108 Molecular & Cell Biology - Cancer & Development
1.108.1327 Ciliopathies
Web Of Science research areas
Veterinary Sciences
ESI research areas
Plant & Animal Science
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