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The effects of MCC-134 on the ATP-sensitive K+ channels in pig urethra
Journal article   Peer reviewed

The effects of MCC-134 on the ATP-sensitive K+ channels in pig urethra

T. Yunoki, N. Teramoto, N. Takano, N. Seki, K.E. Creed, S. Naito and Y. Ito
European Journal of Pharmacology, Vol.482(1-3), pp.287-295
2003
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Abstract

We have investigated the effects of MCC-134 (1-[4-(1H-imidazol-1-yl)benzoyl]-N-methyl-cyclobutanecarbothioamide) on membrane currents and ATP-sensitive K+ channel (KATP channel) opener-induced currents in pig urethra by use of patch-clamp techniques (conventional whole-cell configuration and nystatin perforated patch recordings). Tension measurement was also performed to study the effects of MCC-134 on the resting tone of pig urethral strips. MCC-134 reduced the resting tone of pig urethra in a concentration-dependent manner (EC50=6 μM). The MCC-134 (30 μM)-induced relaxation was suppressed by glibenclamide. In voltage-clamp experiments, MCC-134 produced a concentration-dependent inward K+ current which was suppressed by application of glibenclamide at a holding potential of −50 mV (symmetrical 140 mM K+ conditions). Application of MCC-134 enhanced diazoxide-induced inward currents and inhibited pinacidil-induced inward currents in a concentration-dependent manner at −50 mV. These results suggest that MCC-134 induces glibenclamide-sensitive KATP currents in pig urethra.

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Citation topics
1 Clinical & Life Sciences
1.26 Diabetes
1.26.361 Pancreatic Islet Research
Web Of Science research areas
Pharmacology & Pharmacy
ESI research areas
Pharmacology & Toxicology
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