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The extracellular matrix and mechanotransduction in pulmonary fibrosis
Journal article   Peer reviewed

The extracellular matrix and mechanotransduction in pulmonary fibrosis

Z. Deng, M.W. Fear, Y. Suk Choi, F.M. Wood, A. Allahham, S.E. Mutsaers and C.M. Prêle
The International Journal of Biochemistry & Cell Biology, Vol.126, Art. 105802
2020
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Abstract

Pulmonary fibrosis is characterised by excessive scarring in the lung which leads to compromised lung function, serious breathing problems and in some diseases, death. It includes several lung disorders with idiopathic pulmonary fibrosis (IPF) the most common and most severe. Pulmonary fibrosis is considered to be perpetuated by aberrant wound healing which leads to fibroblast accumulation, differentiation and activation, and deposition of excessive amounts of extracellular matrix (ECM) components, in particular, collagen. Recent studies have identified the importance of changes in the composition and structure of lung ECM during the development of pulmonary fibrosis and the interaction between ECM and lung cells. There is strong evidence that increased matrix stiffness induces changes in cell function including proliferation, migration, differentiation and activation. Understanding how changes in the ECM microenvironment influence cell behaviour during fibrogenesis, and the mechanisms regulating these changes, will provide insight for developing new treatments.

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#3 Good Health and Well-Being

Source: InCites

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Collaboration types
Domestic collaboration
Citation topics
1 Clinical & Life Sciences
1.208 Vasculitis & Autoimmune Disorders
1.208.1262 Idiopathic Pulmonary Fibrosis
Web Of Science research areas
Biochemistry & Molecular Biology
Cell Biology
ESI research areas
Biology & Biochemistry
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